Small Variations Between Species/Subtypes Attributed to Reassortment Evidenced from Polymerase Basic Protein 1 with Other Seven Proteins from Influenza A Virus
Identifieur interne : 000937 ( Main/Exploration ); précédent : 000936; suivant : 000938Small Variations Between Species/Subtypes Attributed to Reassortment Evidenced from Polymerase Basic Protein 1 with Other Seven Proteins from Influenza A Virus
Auteurs : S. Yan [République populaire de Chine] ; G. Wu [République populaire de Chine]Source :
- Transboundary and Emerging Diseases [ 1865-1674 ] ; 2013-04.
English descriptors
- Teeft :
- Anova, Avian, Avian viruses, Binding mechanism, Biochem, Biological functions, Biophys, Blackwell verlag gmbh transboundary, Chou, Commun, Comparison test, Different host species, Different proteins, Different species, Different subtypes, Genetic segments, Gmbh, Host species, Huang, Interaction networks, Large variations, Many studies, Matrix protein, Mutation, Neuraminidase, Numeric feature, Other hand, Other species, Other subtypes, Pielak, Plo, Polymerase, Polymerase acidic protein, Previous studies, Proton channel, Reassortment, Subtypes, Transboundary, Verlag, Virus, Wang.
Abstract
This is a continuation of our studies previously published in this journal to use data analysis to explore why the reassortment of genetic segments from different host species and from different subtypes of influenza A viruses occurred frequently, which was considered the main reason for the generation of new strains. Of eleven proteins from influenza A virus, we have studied seven proteins in our previous studies. To get a full picture, 2352 polymerase basic proteins 1 from influenza A viruses were analysed. The results showed that the variations between host species/subtypes are smaller than those within host species/subtype. In combination with the results obtained from hemagglutinin, neuraminidase, nucleoprotein, matrix proteins 1 and 2, polymerase acidic protein and polymerase basic proteins 1 and 2, the results suggested that the predisposition to reassortment of genetic segments of influenza A virus from different host species and subtypes was mainly because of the small variations between the virus isolates.
Url:
DOI: 10.1111/j.1865-1682.2012.01323.x
Affiliations:
Links toward previous steps (curation, corpus...)
- to stream Istex, to step Corpus: 000781
- to stream Istex, to step Curation: 000781
- to stream Istex, to step Checkpoint: 000047
- to stream Main, to step Merge: 000938
- to stream Main, to step Curation: 000937
Le document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Small Variations Between Species/Subtypes Attributed to Reassortment Evidenced from Polymerase Basic Protein 1 with Other Seven Proteins from Influenza A Virus</title><author><name sortKey="Yan, S" sort="Yan, S" uniqKey="Yan S" first="S." last="Yan">S. Yan</name></author><author><name sortKey="Wu, G" sort="Wu, G" uniqKey="Wu G" first="G." last="Wu">G. Wu</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:AF02F20B335B7AA4083E6702EFF479F98A1625E6</idno><date when="2013" year="2013">2013</date><idno type="doi">10.1111/j.1865-1682.2012.01323.x</idno><idno type="url">https://api.istex.fr/ark:/67375/WNG-SS2RFQ6D-Z/fulltext.pdf</idno><idno type="wicri:Area/Istex/Corpus">000781</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">000781</idno><idno type="wicri:Area/Istex/Curation">000781</idno><idno type="wicri:Area/Istex/Checkpoint">000047</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Checkpoint">000047</idno><idno type="wicri:doubleKey">1865-1674:2013:Yan S:small:variations:between</idno><idno type="wicri:Area/Main/Merge">000938</idno><idno type="wicri:Area/Main/Curation">000937</idno><idno type="wicri:Area/Main/Exploration">000937</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main">Small Variations Between Species/Subtypes Attributed to Reassortment Evidenced from Polymerase Basic Protein 1 with Other Seven Proteins from Influenza A Virus</title><author><name sortKey="Yan, S" sort="Yan, S" uniqKey="Yan S" first="S." last="Yan">S. Yan</name><affiliation wicri:level="1"><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea> State Key Laboratory of Non‐food Biomass Enzyme Technology, National Engineering Research Center for Non‐food Biorefinery, Guangxi Key Laboratory of Biorefinery, Guangxi Academy of Sciences, Nanning</wicri:regionArea><wicri:noRegion>Nanning</wicri:noRegion></affiliation></author><author><name sortKey="Wu, G" sort="Wu, G" uniqKey="Wu G" first="G." last="Wu">G. Wu</name><affiliation wicri:level="1"><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea> State Key Laboratory of Non‐food Biomass Enzyme Technology, National Engineering Research Center for Non‐food Biorefinery, Guangxi Key Laboratory of Biorefinery, Guangxi Academy of Sciences, Nanning</wicri:regionArea><wicri:noRegion>Nanning</wicri:noRegion></affiliation><affiliation wicri:level="3"><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea> DreamSciTech Consulting, Shenzhen</wicri:regionArea><placeName><settlement type="city">Shenzhen</settlement><region type="province">Guangdong</region></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">Transboundary and Emerging Diseases</title><title level="j" type="alt">TRANSBOUNDARY AND EMERGING DISEASES</title><idno type="ISSN">1865-1674</idno><idno type="eISSN">1865-1682</idno><imprint><biblScope unit="vol">60</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="110">110</biblScope><biblScope unit="page" to="119">119</biblScope><biblScope unit="page-count">10</biblScope><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2013-04">2013-04</date></imprint><idno type="ISSN">1865-1674</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1865-1674</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Anova</term><term>Avian</term><term>Avian viruses</term><term>Binding mechanism</term><term>Biochem</term><term>Biological functions</term><term>Biophys</term><term>Blackwell verlag gmbh transboundary</term><term>Chou</term><term>Commun</term><term>Comparison test</term><term>Different host species</term><term>Different proteins</term><term>Different species</term><term>Different subtypes</term><term>Genetic segments</term><term>Gmbh</term><term>Host species</term><term>Huang</term><term>Interaction networks</term><term>Large variations</term><term>Many studies</term><term>Matrix protein</term><term>Mutation</term><term>Neuraminidase</term><term>Numeric feature</term><term>Other hand</term><term>Other species</term><term>Other subtypes</term><term>Pielak</term><term>Plo</term><term>Polymerase</term><term>Polymerase acidic protein</term><term>Previous studies</term><term>Proton channel</term><term>Reassortment</term><term>Subtypes</term><term>Transboundary</term><term>Verlag</term><term>Virus</term><term>Wang</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">This is a continuation of our studies previously published in this journal to use data analysis to explore why the reassortment of genetic segments from different host species and from different subtypes of influenza A viruses occurred frequently, which was considered the main reason for the generation of new strains. Of eleven proteins from influenza A virus, we have studied seven proteins in our previous studies. To get a full picture, 2352 polymerase basic proteins 1 from influenza A viruses were analysed. The results showed that the variations between host species/subtypes are smaller than those within host species/subtype. In combination with the results obtained from hemagglutinin, neuraminidase, nucleoprotein, matrix proteins 1 and 2, polymerase acidic protein and polymerase basic proteins 1 and 2, the results suggested that the predisposition to reassortment of genetic segments of influenza A virus from different host species and subtypes was mainly because of the small variations between the virus isolates.</div></front></TEI><affiliations><list><country><li>République populaire de Chine</li></country><region><li>Guangdong</li></region><settlement><li>Shenzhen</li></settlement></list><tree><country name="République populaire de Chine"><noRegion><name sortKey="Yan, S" sort="Yan, S" uniqKey="Yan S" first="S." last="Yan">S. Yan</name></noRegion><name sortKey="Wu, G" sort="Wu, G" uniqKey="Wu G" first="G." last="Wu">G. Wu</name><name sortKey="Wu, G" sort="Wu, G" uniqKey="Wu G" first="G." last="Wu">G. Wu</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Sante/explor/H2N2V1/Data/Main/Exploration
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 000937 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd -nk 000937 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien
|wiki= Sante
|area= H2N2V1
|flux= Main
|étape= Exploration
|type= RBID
|clé= ISTEX:AF02F20B335B7AA4083E6702EFF479F98A1625E6
|texte= Small Variations Between Species/Subtypes Attributed to Reassortment Evidenced from Polymerase Basic Protein 1 with Other Seven Proteins from Influenza A Virus
}}
| This area was generated with Dilib version V0.6.33. |  |